In Adaptive Immunity, Receptors Provide Pathogen-Specific Recognition

Rucete ✏ Campbell Biology In a Nutshell

Unit 7 ANIMAL FORM AND FUNCTION — Concept 43.2 In Adaptive Immunity, Receptors Provide Pathogen-Specific Recognition

Adaptive immunity, found only in vertebrates, relies on highly specific receptors on B and T cells that recognize particular molecules on pathogens. It develops more slowly than innate immunity but provides stronger and faster responses upon re-exposure due to immunological memory.

1. Antigens and Lymphocytes

  • An antigen is any molecule that elicits a response from B or T cells
  • Each lymphocyte (B or T cell) has receptors specific to one antigen epitope
  • B cells mature in bone marrow; T cells mature in thymus
  • A single B or T cell has ~100,000 identical antigen receptors

2. Antigen Receptors

  • B cell receptors: Y-shaped proteins with 2 identical binding sites
  • Binding leads to secretion of antibodies (immunoglobulins), which neutralize pathogens
  • Antibodies recognize intact antigens in body fluids or on pathogen surfaces
  • T cell receptors: bind to antigen fragments presented by MHC molecules on host cells
  • Class I MHC: on all nucleated cells; Class II MHC: on antigen-presenting cells

3. Diversity of Receptors

  • Immense diversity (~1 million B cell types, 10 million T cell types) arises from gene recombination
  • V (variable), J (joining), and C (constant) gene segments combine randomly
  • Recombinase enzyme joins segments during lymphocyte development
  • Each receptor is unique and permanent once rearranged

4. Self-Tolerance

  • Developing lymphocytes are tested for self-reactivity
  • Those that bind self-molecules are either eliminated by apoptosis or inactivated
  • Ensures immune system does not attack its own cells

5. Clonal Selection and Proliferation

  • When a receptor binds its specific antigen, the cell proliferates into a clone
  • Clone includes effector cells (e.g., plasma cells, helper T cells) and memory cells
  • This process is called clonal selection

6. Immunological Memory

  • Primary response: slower, begins ~10–17 days after first exposure
  • Secondary response: faster, stronger, and longer due to memory cells (2–7 days)
  • Memory cells persist for decades and respond rapidly upon re-infection

In a Nutshell

Adaptive immunity uses millions of unique receptors on B and T cells to recognize specific pathogen components. It includes antigen recognition, clonal expansion, and memory formation. The result is a targeted, efficient defense that improves with repeated exposure, forming the basis for long-term immunity.

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