Adaptive Immunity Defends against Infection of Body Fluids and Body Cells

Rucete ✏ Campbell Biology In a Nutshell

Unit 7 ANIMAL FORM AND FUNCTION — Concept 43.3 Adaptive Immunity Defends against Infection of Body Fluids and Body Cells

The adaptive immune response is carried out by B and T lymphocytes, which work together to fight pathogens in body fluids and inside infected cells. Helper T cells play a central coordinating role in activating both humoral (antibody-mediated) and cell-mediated responses.

1. Helper T Cells Activate Immune Responses

  • Helper T cells are activated when they bind to an antigen presented by a class II MHC on an antigen-presenting cell (macrophage, dendritic cell, or B cell)
  • Binding is stabilized by CD4, an accessory protein
  • Activated helper T cells release cytokines that stimulate B cells and cytotoxic T cells
  • Helper T cells are essential for both humoral and cell-mediated responses

2. B Cells and Humoral Immunity

  • B cells internalize antigens via receptor-mediated endocytosis and present fragments with class II MHC
  • Activated helper T cells bind and activate B cells with the same antigen specificity
  • Activated B cells proliferate and differentiate into plasma cells (secreting antibodies) and memory B cells
  • Plasma cells secrete ~2,000 antibodies per second for 4–5 days
  • Each exposure activates multiple B cells due to multiple epitopes on antigens

3. Antibody Functions

  • Neutralization: Antibodies block virus or toxin entry into cells
  • Opsonization: Antibodies enhance phagocytosis by linking pathogens into clumps
  • Antibodies activate complement proteins, leading to membrane attack complex and cell lysis
  • Some antibodies recruit natural killer (NK) cells to destroy infected cells
  • Five antibody classes (IgA, IgD, IgE, IgG, IgM) differ in constant region but have same specificity

4. Cytotoxic T Cells and Cell-Mediated Immunity

  • Cytotoxic T cells recognize infected cells via class I MHC presenting foreign antigen fragments
  • Stabilized by CD8 protein
  • Once activated, they release perforin and granzymes that trigger apoptosis in infected cells
  • This destroys the infection site and exposes pathogen to antibody action

5. Summary of Adaptive Immunity

  • Humoral immunity defends against pathogens in blood and lymph using secreted antibodies
  • Cell-mediated immunity targets infected cells and cancer cells with cytotoxic T cells
  • Both systems rely on memory cells for faster secondary responses upon re-exposure

6. Active and Passive Immunity

  • Active immunity develops from infection or vaccination and produces memory
  • Passive immunity comes from receiving antibodies (e.g. from mother via placenta or breast milk, or injected antivenin)
  • Passive immunity is immediate but temporary

7. Immunization

  • Vaccines stimulate primary responses and memory cell formation
  • Secondary exposure leads to rapid, strong secondary responses
  • Widespread vaccination has eradicated smallpox and reduced diseases like polio and measles
  • Misinformation about vaccines has caused dangerous declines in immunization rates

8. Immune Rejection and Antibodies as Tools

  • Transplants are rejected due to MHC mismatches
  • Monoclonal antibodies, made from a single B cell clone, are used for diagnostics and therapy
  • New technologies can identify all viruses a person has been exposed to using antibody profiling

In a Nutshell

The adaptive immune system uses helper T cells to coordinate humoral and cell-mediated responses. B cells produce antibodies to neutralize extracellular threats, while cytotoxic T cells kill infected host cells. Vaccines enhance immunity by generating memory cells. Through clonal selection, diversity, and memory, adaptive immunity provides precise, long-lasting protection tailored to each pathogen.

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